Fefrillas have identified a protein that helps prevent excessive fright during wound healing. The protein, called fibromodulin (fmoda), forms a complex of molecules with interleukine 1β that prevents myofibroblasts from forming excessive scar tissue.
The findings can lead to a new way to reduce or prevent excess scars, which could benefit from patterns recovering from surgery, insjuries or burns. The study is published in Nature communication.
In earlier studies, the researchers had described the crucial role of FMOD in strengthening scarless fetal-type repair and preventing excessive fright during the repair of the adult type. For this new study, research is investigating the effects of FMOD on Myofibroblats, into an important cell in healing and scarring. Myofibroblasts have to die, one na -wound healings, but they can continue to form tissue long after, which leads to excessive scars. The complex -based complex promotes the death of Myofibroblats.
“Dr. Eric Kang Ting and I have studied how the skin shift can be reduced at UCLA in the past two decades,” says Chia Soo, MD, professor surgery and vice -chairman for Reesar for the UCLA division of plastic and reconstructive surgery, in release. “This Publication Explores The Crucial Role of Naturally Occurring Fibromodulin in Regulating a Key Scar-Forming Cell, The Myofibroblast. This, Combined with Team’s fda-approved Clinical Study Led by Dr Zhong Pattery-Derive-Derive-Der-Der-Der-Der-Der-Der-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive-Derive Scar Reduction, Demonstrates A Significant Leap in Potential Treatments for Patients with Excessive Scarring.
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